Proceedings of the ... International Congress of Human Genetics, Band 3S. Karger, 1967 - 578 Seiten |
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Seite 173
... gene Se may be considered as a regulator , or switch , gene that controls the expression of the H gene , and the genes O , h , se , and le as silent alleles , i.e. , as genes that do not give products which control structural changes in ...
... gene Se may be considered as a regulator , or switch , gene that controls the expression of the H gene , and the genes O , h , se , and le as silent alleles , i.e. , as genes that do not give products which control structural changes in ...
Seite 185
... gene can only take place if the individual has a secretor Se gene . Therefore , in the absence of a secretor gene , H , A , and B structures cannot be formed even if the individual has H , A , and B genes . The Le gene is not under the ...
... gene can only take place if the individual has a secretor Se gene . Therefore , in the absence of a secretor gene , H , A , and B structures cannot be formed even if the individual has H , A , and B genes . The Le gene is not under the ...
Seite 320
... genes in different species and to correlate gene duplications with steps in speciation . Presumably the evolution of the hemoglobin and myoglobin genes has followed a similar , but not identical , pattern in different species . The ...
... genes in different species and to correlate gene duplications with steps in speciation . Presumably the evolution of the hemoglobin and myoglobin genes has followed a similar , but not identical , pattern in different species . The ...
Inhalt
Welcoming Address | 3 |
Remarks of the Secretary of the Permanent Committee for the International | 11 |
Diagnosis in medical genetics | 29 |
Urheberrecht | |
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abnormal acatalasia acatalatic acid Acta activity alleles Amer amino-acid amino-acid sequence analysis aneuploidy anomalies antibodies antigenic autosomal dominant autosomal recessive biochemical birth blood blood-group catalase cent child chromosome aberrations clinical colonies congenital consanguinity Cytogenetics defect deficiency Dept determined disaccharidases disease distribution duplication effects enzyme evolution factors female gamma globulin gene frequencies hemoglobin heredity is autosomal heterochromatin heterozygotes heterozygous Hospital Human Genetics inactivation individuals inheritance intestinal karyotype L-leut lactase Lancet leukemia linkage loci locus male malformations maltase marker marriage mating Medical mental metabolism Michigan Mongolism mosaic mutation normal observed pairs parents patients pattern pedigree peptide chains Ph¹ phenotype population possible Proc protein recombination records red cells selection sex chromatin sex-linked specific structure studies substance sucrase syndrome synthesis Table techniques tion tissue traits translocation trisomy University variable variation X-chromosome York