Génétique HumaineJean de Grouchy, Francis John Govier Ebling, I. W. Henderson Excerpta Medica, 1972 - 499 Seiten |
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Seite 85
... Table 2 , is appropriate . The second assumption ( no cross - migration ) is probably correct since groups far apart are being consider- ed . The ƒ value on these assumptions is related to the time t ( in generations ) since the groups ...
... Table 2 , is appropriate . The second assumption ( no cross - migration ) is probably correct since groups far apart are being consider- ed . The ƒ value on these assumptions is related to the time t ( in generations ) since the groups ...
Seite 157
... Table 2. As can be seen , the index rose to a maximum for the cohort that was age 25-29 in 1930 to 1935. This corresponds to a period of low birth rate . As with the Chilean data , a reduction of fertility was accompanied by a rise in ...
... Table 2. As can be seen , the index rose to a maximum for the cohort that was age 25-29 in 1930 to 1935. This corresponds to a period of low birth rate . As with the Chilean data , a reduction of fertility was accompanied by a rise in ...
Seite 451
... TABLE 6 Position in sibship of recombinants for the Lutheran / Secretor and Lutheran / myotonic dystrophy linkage pairs Maternal Paternal First half Second half 1 0 2 8 The results for 10 families informative for the Lutheran / Secretor ...
... TABLE 6 Position in sibship of recombinants for the Lutheran / Secretor and Lutheran / myotonic dystrophy linkage pairs Maternal Paternal First half Second half 1 0 2 8 The results for 10 families informative for the Lutheran / Secretor ...
Inhalt
Clinical geneticsGénétique clinique | 25 |
Radiation geneticsRadiations ionisantes | 38 |
Données récentes sur les taux de mutations radioinduites chez | 67 |
Urheberrecht | |
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abnormal Acad acid activity alleles analysis aneuploid anti anticoagulant anticorps antigen antipyrine autosomal biochemical bishydroxycoumarin blood cell hybrids cell lines chimerism clinical clones culture Cytogenetics deficiency dehydrogenase detected deux diagnosis dose drug metabolism effect electrophoretic enzyme assay estimate été être factors Fagerhol female fibroblasts fraction G6PD gamètes gène genetic genetic control génétique hereditary heterozygotes heterozygous hexosaminidase HL-A human chromosomes individuals inheritance isozymes karyotype Level linkage lipoprotein loci locus Lp(a Lp(a+ lymphocytes lymphoid cell male marker meiosis meiotic mental retardation mouse mutation Nature Lond Negro normal observed pairs parents patients patterns peut pharmacogenetics phenotype phenylbutazone plasma polymorphism population possible Proc produit propositus protein prothrombin Pygmies rearrangement recombination red cells sera serum sibship singletons somatic cell structure studies syndrome Table Tay-Sachs disease tests tion tissues translocations twins variants Vesell vitamin vitamin K warfarin X chromosome XX/XY