Génétique HumaineJean de Grouchy, Francis John Govier Ebling, I. W. Henderson Excerpta Medica, 1972 - 499 Seiten |
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Seite 183
... appears to be very different from the others , that is the specific lesions caused in chromosome number 17 by the adminis- tration of adenovirus 12. This was first reported by Zur Hausen ( 1967 ) and has recently been confirmed in my ...
... appears to be very different from the others , that is the specific lesions caused in chromosome number 17 by the adminis- tration of adenovirus 12. This was first reported by Zur Hausen ( 1967 ) and has recently been confirmed in my ...
Seite 197
... appears as a prominent dense chromomere , almost always a single structure . The 5 acrocentric bivalents are relatively easily identified by their large terminal nucleolar chromomeres and their association with nucleoli . There are ...
... appears as a prominent dense chromomere , almost always a single structure . The 5 acrocentric bivalents are relatively easily identified by their large terminal nucleolar chromomeres and their association with nucleoli . There are ...
Seite 274
... appears that for any given gene locus coding for the structure of a particular enzyme or protein a series of different mutant alleles each resulting in marked functional deficiency and hence in a characteristic clinical abnormality may ...
... appears that for any given gene locus coding for the structure of a particular enzyme or protein a series of different mutant alleles each resulting in marked functional deficiency and hence in a characteristic clinical abnormality may ...
Inhalt
Clinical geneticsGénétique clinique | 25 |
Radiation geneticsRadiations ionisantes | 38 |
Données récentes sur les taux de mutations radioinduites chez | 67 |
Urheberrecht | |
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abnormal Acad acid activity alleles analysis aneuploid anti anticoagulant anticorps antigen antipyrine autosomal biochemical bishydroxycoumarin blood cell hybrids cell lines chimerism clinical clones culture Cytogenetics deficiency dehydrogenase detected deux diagnosis dose drug metabolism effect electrophoretic enzyme assay estimate été être factors Fagerhol female fibroblasts fraction G6PD gamètes gène genetic genetic control génétique hereditary heterozygotes heterozygous hexosaminidase HL-A human chromosomes individuals inheritance isozymes karyotype Level linkage lipoprotein loci locus Lp(a Lp(a+ lymphocytes lymphoid cell male marker meiosis meiotic mental retardation mouse mutation Nature Lond Negro normal observed pairs parents patients patterns peut pharmacogenetics phenotype phenylbutazone plasma polymorphism population possible Proc produit propositus protein prothrombin Pygmies rearrangement recombination red cells sera serum sibship singletons somatic cell structure studies syndrome Table Tay-Sachs disease tests tion tissues translocations twins variants Vesell vitamin vitamin K warfarin X chromosome XX/XY