Génétique HumaineJean de Grouchy, Francis John Govier Ebling, I. W. Henderson Excerpta Medica, 1972 - 499 Seiten |
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Seite 6
... determined disease into greater prominence . The increased survival rate of patients with such disorders , putting a long - term strain on the parents and the community ; for example , in England 40 years ago 7 out of 8 children with ...
... determined disease into greater prominence . The increased survival rate of patients with such disorders , putting a long - term strain on the parents and the community ; for example , in England 40 years ago 7 out of 8 children with ...
Seite 18
... Determined by expressing linear growth as : C. Stewart . ; the normal rate being taken from Harvard growth chart of Harold cm / yr at 50th percentile for controls *** Determined by Dr. Anne Roseborough upon heparinized blood specimens ...
... Determined by expressing linear growth as : C. Stewart . ; the normal rate being taken from Harvard growth chart of Harold cm / yr at 50th percentile for controls *** Determined by Dr. Anne Roseborough upon heparinized blood specimens ...
Seite 386
... determined heritability ( Schaefer et al . , 1958 ) . Some may be affected with the autosomal dominant type II hypercholesterolemia ( Levy and Langer , 1971 ) and others with a more poorly defined separate genetic disorder associated ...
... determined heritability ( Schaefer et al . , 1958 ) . Some may be affected with the autosomal dominant type II hypercholesterolemia ( Levy and Langer , 1971 ) and others with a more poorly defined separate genetic disorder associated ...
Inhalt
Clinical geneticsGénétique clinique | 25 |
Radiation geneticsRadiations ionisantes | 38 |
Données récentes sur les taux de mutations radioinduites chez | 67 |
Urheberrecht | |
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abnormal Acad acid activity alleles analysis aneuploid anti anticoagulant anticorps antigen antipyrine autosomal biochemical bishydroxycoumarin blood cell hybrids cell lines chimerism clinical clones culture Cytogenetics deficiency dehydrogenase detected deux diagnosis dose drug metabolism effect electrophoretic enzyme assay estimate été être factors Fagerhol female fibroblasts fraction G6PD gamètes gène genetic genetic control génétique hereditary heterozygotes heterozygous hexosaminidase HL-A human chromosomes individuals inheritance isozymes karyotype Level linkage lipoprotein loci locus Lp(a Lp(a+ lymphocytes lymphoid cell male marker meiosis meiotic mental retardation mouse mutation Nature Lond Negro normal observed pairs parents patients patterns peut pharmacogenetics phenotype phenylbutazone plasma polymorphism population possible Proc produit propositus protein prothrombin Pygmies rearrangement recombination red cells sera serum sibship singletons somatic cell structure studies syndrome Table Tay-Sachs disease tests tion tissues translocations twins variants Vesell vitamin vitamin K warfarin X chromosome XX/XY