Génétique HumaineJean de Grouchy, Francis John Govier Ebling, I. W. Henderson Excerpta Medica, 1972 - 499 Seiten |
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Seite 85
... major cause of genetic variation in the polymorphisms considered ; or , more generally , that , if selection also is present , its disruptive and homeostatic effects , which would respectively increase and lower the ƒ values , tend to ...
... major cause of genetic variation in the polymorphisms considered ; or , more generally , that , if selection also is present , its disruptive and homeostatic effects , which would respectively increase and lower the ƒ values , tend to ...
Seite 105
... major complex ( details in Ward and Neel , 1970 ) . Figure 5 is our best genetic network for these same 7 villages , based on gene frequencies at 11 loci ( Rh , MNSs , Duffy , Kidd , Diego , P , Lewis , haptoglobin , Gc , acid ...
... major complex ( details in Ward and Neel , 1970 ) . Figure 5 is our best genetic network for these same 7 villages , based on gene frequencies at 11 loci ( Rh , MNSs , Duffy , Kidd , Diego , P , Lewis , haptoglobin , Gc , acid ...
Seite 163
... major inbreeding component is major genes . There is no significant non - linearity at low doses ; in fact there is ordinarily no measurable minor component . The average person is heterozygous for some 2-3 lethal equivalents , the bulk ...
... major inbreeding component is major genes . There is no significant non - linearity at low doses ; in fact there is ordinarily no measurable minor component . The average person is heterozygous for some 2-3 lethal equivalents , the bulk ...
Inhalt
Clinical geneticsGénétique clinique | 25 |
Radiation geneticsRadiations ionisantes | 38 |
Données récentes sur les taux de mutations radioinduites chez | 67 |
Urheberrecht | |
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abnormal Acad acid activity alleles analysis aneuploid anti anticoagulant anticorps antigen antipyrine autosomal biochemical bishydroxycoumarin blood cell hybrids cell lines chimerism clinical clones culture Cytogenetics deficiency dehydrogenase detected deux diagnosis dose drug metabolism effect electrophoretic enzyme assay estimate été être factors Fagerhol female fibroblasts fraction G6PD gamètes gène genetic genetic control génétique hereditary heterozygotes heterozygous hexosaminidase HL-A human chromosomes individuals inheritance isozymes karyotype Level linkage lipoprotein loci locus Lp(a Lp(a+ lymphocytes lymphoid cell male marker meiosis meiotic mental retardation mouse mutation Nature Lond Negro normal observed pairs parents patients patterns peut pharmacogenetics phenotype phenylbutazone plasma polymorphism population possible Proc produit propositus protein prothrombin Pygmies rearrangement recombination red cells sera serum sibship singletons somatic cell structure studies syndrome Table Tay-Sachs disease tests tion tissues translocations twins variants Vesell vitamin vitamin K warfarin X chromosome XX/XY