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Case 1 Lymphocytes Fibroblasts Case 2 Lymphocytes Fibroblasts Case 3 Lymphocytes Case 4 Lymphocytes B Case 5 Lymphocytes Chromosome count -44 74 4 10 5 2 3 45 33 2 46 288 10 175 2 15 4 97 52 1 84 146 47 11 Total 107 35 2 197 22 54 1 88 ...
Case 1 Lymphocytes Fibroblasts Case 2 Lymphocytes Fibroblasts Case 3 Lymphocytes Case 4 Lymphocytes B Case 5 Lymphocytes Chromosome count -44 74 4 10 5 2 3 45 33 2 46 288 10 175 2 15 4 97 52 1 84 146 47 11 Total 107 35 2 197 22 54 1 88 ...
Seite 300
The work of Doherty and Zinkernagel ( 1975 ) has recently provided striking evidence for a role for these universal serological determinants in recognition by immune T lymphocytes of their specific targets ( see also Zinkernagel ...
The work of Doherty and Zinkernagel ( 1975 ) has recently provided striking evidence for a role for these universal serological determinants in recognition by immune T lymphocytes of their specific targets ( see also Zinkernagel ...
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THE CELLOLOGY OF LYMPHOCYTE RESPONSES TO CELL MEMBRANE STRUCTURES CODED FOR IN HLA AND NON - HLA REGIONS * B. A. ... and effector stages were represented in part by the specific ability of sensitized lymphocytes to kill target cells .
THE CELLOLOGY OF LYMPHOCYTE RESPONSES TO CELL MEMBRANE STRUCTURES CODED FOR IN HLA AND NON - HLA REGIONS * B. A. ... and effector stages were represented in part by the specific ability of sensitized lymphocytes to kill target cells .
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The legal aspects of genetic screening and counseling | 3 |
Population genetics R C Lewontin | 13 |
Some trends in the study of spontaneous and induced mutation in | 19 |
Urheberrecht | |
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abnormalities activity addition agents alleles amniocentesis analysis antigens appears approach associated blood cancer cause cells chemical chromosome chromosome abnormalities Class common complex culture damage defects deficiency demonstrated determined diagnosis disease disorders distance Down's syndrome Editors effects environmental enzyme estimates et al evidence example expressed factors frequency function gene genetic groups human hybrids important incidence increased indicate individuals induced inheritance involved known lactose least less levels lines loci locus lymphocytes major maternal measures mechanisms methods mouse mutation Nature neural tube normal observed obtained occur origin parents patients phenotypes polymorphism population possible prenatal present Press probably problem protein recent REFERENCES region relatively repair reported response risk samples screening selection sensitivity shown single somatic specific structure studies suggested syndrome Table techniques trisomy tumors variants